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Cardiology

Chest Pain Assessment and Acute Coronary Syndrome

Understanding Chest Pain Etiology

Chest pain can originate from multiple anatomical structures. A systematic approach by anatomical layer helps narrow the differential diagnosis:

Layer Common Conditions
Skin & Subcutaneous Laceration, herpes zoster, cellulitis
Musculoskeletal Costochondritis, muscle strain, rib fractures
Pleural Pleurisy, pneumothorax, pneumonia
Pericardium Pericarditis, pericardial effusion, tamponade
Cardiac Acute coronary syndrome, myocarditis, acute heart failure
Esophageal Gastroesophageal reflux disease, esophageal spasm, perforation
Aortic Aortic dissection, thoracic aortic aneurysm
Abdominal Biliary colic, pancreatitis, peptic ulcer disease
Psychiatric Anxiety disorder, panic attacks

Angina Classification Systems

Diamond Classification (Typicality of Angina)

The Diamond classification helps stratify the probability that chest pain represents true anginal discomfort:

  • 3 of 3 typical features: Substernal location + Provoked by exertion + Relieved by nitroglycerin or rest = Typical angina
  • 2 of 3 typical features = Atypical angina
  • 1 of 3 typical features = Non-anginal chest pain

Clinical Pearl

Use this classification to guide your pretest probability assessment before ordering additional diagnostic testing.

Canadian Cardiovascular Society Grading

This scale quantifies functional limitations from angina:

Grade Characteristics
I Chest pain only with strenuous or prolonged exertion
II Mild limitation; pain with light exertion (climbing stairs, walking >2 blocks)
III Marked limitation; pain with ordinary activities (walking 1-2 blocks on flat surface)
IV Unable to perform any activity without symptoms; pain at rest

Initial Assessment and Workup

Time-Critical Actions

All chest pain patients require an EKG within 10 minutes of arrival.

Immediate Diagnostic Tests

  1. Electrocardiogram — obtain within 10 minutes
  2. Complete blood count — assess for anemia or infection
  3. Comprehensive metabolic panel — electrolytes, renal function, glucose
  4. Oxygen saturation — by pulse oximetry
  5. Chest radiograph — assess for alternative diagnoses
  6. Cardiac telemetry — continuous monitoring for arrhythmias
  7. Initial troponin — high-sensitivity troponin preferred

Serial Testing Protocol

  • Repeat EKG at 2-3 minutes if initial is normal or nondiagnostic and clinical suspicion remains high
  • Serial troponin measurements at 4-6 hour intervals for at least 2 measurements
  • Troponin may be normal in the first 3 hours of symptom onset

EKG Localization of Myocardial Infarction

Understanding which coronary artery corresponds to EKG changes guides your assessment:

EKG Leads Myocardial Region Likely Culprit Artery
V1–V2 Septal wall Left anterior descending
V3–V4 Anterior wall Left anterior descending
I, aVL Lateral wall Left circumflex or LCx diagonal
V5–V6 Lateral wall Left circumflex
II, III, aVF Inferior wall Right coronary artery
V4R Right ventricle Right coronary artery
V1–V3 (ST depression) Posterior wall (reciprocal) Right coronary or left circumflex

RV Infarction Clue

If inferior STEMI is present, always check lead V4R for ST elevation suggesting right ventricular involvement.

Risk Stratification Scores

TIMI Risk Score for Unstable Angina/NSTEMI

Calculate points (0-7 total) and correlate with mortality risk:

Criterion Points
Age ≥65 years 1
≥3 cardiac risk factors 1
Known CAD (stenosis ≥50%) 1
Aspirin use in past 7 days 1
ST-segment deviation ≥0.5 mm 1
Elevated cardiac biomarkers 1
Severe angina (≥2 episodes in 24h) 1

Risk stratification: 0-1 points = low risk, 2-4 points = intermediate risk, 5-7 points = high risk

GRACE Risk Score

The GRACE score incorporates multiple variables to predict in-hospital and 6-month mortality. Consult scoring tools/nomograms for accurate calculation. Generally:

  • Score <140 = Low risk
  • Score 140–200 = Intermediate risk
  • Score >200 = High risk

Understanding Troponin Elevation

Elevated cardiac troponin indicates myocardial injury but does not specify etiology:

Type Mechanism Examples
Type 1 Atherosclerotic plaque rupture with thrombosis STEMI, NSTEMI
Type 2 Myocardial supply-demand mismatch Sepsis, anemia, hypertensive crisis, tachyarrhythmia, hypoxia
Non-MI elevation Other myocardial processes Heart failure, myocarditis, renal failure, pulmonary embolism, sepsis

Critical Insight

Elevated troponin requires correlation with clinical context, EKG findings, and imaging to determine true MI versus other causes of myocardial injury.

Stress Testing Modalities

Test Type Indications Limitations
Exercise stress test Intermediate pretest probability, interpretable EKG, able to exercise Cannot perform if unable to exercise; unreliable with baseline EKG abnormalities
Pharmacologic stress (adenosine/regadenoson) Unable to exercise, need imaging component Contraindicated in severe asthma/COPD (adenosine)
Dobutamine stress echo Unable to exercise, need structural assessment Induces demand ischemia; contraindicated in uncontrolled HTN or tachyarrhythmia
Coronary CTA Low-intermediate pretest probability, assess for alternative diagnosis Radiation exposure, contrast allergy risk, poor image quality with high heart rate

Acute Coronary Syndrome Treatment

STEMI Protocol

Time is Myocardium

Door-to-balloon time goal: <90 minutes for primary percutaneous coronary intervention.

Immediate management:

  • Aspirin — 325 mg (loading), then 81 mg daily indefinitely
  • Anticoagulation — unfractionated heparin IV bolus/drip or enoxaparin 0.5 mg/kg IV bolus
  • Beta-blocker — if HR >50 and SBP >90 mmHg
  • Nitroglycerin — 0.4 mg sublingual, repeat every 5 minutes as needed for symptom relief
  • Primary PCI — mechanical revascularization within 90 minutes, or
  • Fibrinolysis — if PCI unavailable; initiate within 30 minutes (thrombolytic agents: alteplase, reteplase, tenecteplase)

NSTEMI/Unstable Angina Treatment

Medical management:

  • Aspirin — 325 mg loading dose, then 81 mg daily
  • P2Y12 inhibitor — clopidogrel 600 mg loading (then 75 mg daily), prasugrel, or ticagrelor
  • Anticoagulation — unfractionated heparin drip or LMWH (enoxaparin 1 mg/kg SQ BID)
  • Oxygen — only if SaO2 <90%
  • Nitroglycerin — 0.4 mg sublingual every 5 minutes, transition to IV infusion if ongoing symptoms
  • Beta-blocker — (hold if HR <55 or SBP <100)
  • High-intensity statin — atorvastatin 80 mg daily or rosuvastatin 40 mg daily
  • ACE inhibitor/ARB — initiate at 1-2 days post-infarction if tolerated

Invasive Strategy

Most NSTEMI patients benefit from early invasive evaluation (cardiac catheterization within 12-72 hours depending on risk stratification).


Heart Failure: Classification and Management

Heart Failure Types and Definitions

Heart failure is classified along multiple dimensions:

Ejection Fraction-Based Classification

Classification EF Range Pathophysiology
HFrEF (reduced) <40% Systolic dysfunction; dilated ventricle with poor contractility
HFmrEF (mildly reduced) 40–49% Intermediate systolic function
HFpEF (preserved) ≥50% Diastolic dysfunction; normal EF but impaired filling

Structural Classification

  • Systolic HF: Decreased contractility; EF typically <40%
  • Diastolic HF: Preserved contractility; impaired relaxation or increased stiffness

Output Classification

  • Forward failure: Inadequate perfusion to organs (low cardiac output)
  • Backward failure: Venous congestion upstream of failing ventricle
  • High-output failure: Excessive metabolic demand (sepsis, thyrotoxicosis, anemia, pregnancy)

NYHA Functional Classification and Treatment Strategy

Class Description Symptoms Baseline Treatment
I Asymptomatic No limitation with ordinary activity ACE-I/ARB + Beta-blocker
II Mild symptoms Limitation with strenuous activity + Loop diuretic PRN
III Moderate symptoms Limitation with ordinary activity + Aldosterone antagonist or ISDN/hydralazine combo
IV Severe symptoms Symptoms at rest or with minimal activity Palliative care consideration; cardiac transplant evaluation

Warm-Cold and Wet-Dry Classification

This classification helps categorize hemodynamic status and guides therapy:

cardiac-2 diagram

Common Precipitants of Acute Decompensation

Remember "ADHF" — Acute Decompensated Heart Failure precipitants:

  • Adherenc issues (medication or dietary nonadherence)
  • Dietary indiscretion (excessive sodium or fluid intake)
  • Ischemia (acute MI triggering acute failure)
  • Arrhythmias (new-onset atrial fibrillation or other dysrhythmia)
  • Hypertensive crisis (acute elevation in afterload)
  • Renal failure (reduced diuretic efficacy, volume retention)
  • Fluid overload from other sources (transfusions, IV medications)
  • Infection (pneumonia, UTI, other systemic infection)
  • Endocrine (thyroiditis, hyperthyroidism)

Additionally: Use of NSAIDs, cocaine, discontinuation of guideline-directed medical therapy

Diagnostic Workup

Essential labs and imaging for all decompensated HF:

Test Utility
CBC Assess for anemia (may worsen HF), infection
CMP Electrolytes, renal function, glucose
BNP or NT-proBNP <300 pg/mL excludes HF; >900 makes HF likely; 300–900 indeterminate
TSH Rule out thyroid dysfunction as reversible cause
EKG Assess for ischemia, arrhythmia, chamber enlargement
Chest X-ray Evaluate pulmonary edema, cardiomegaly, alternative diagnoses
Transthoracic echocardiogram Assess EF, diastolic function, valve pathology, RV function

Management by Hemodynamic Category

Universal Measures (All Patients)

  • Sodium restriction: <2 grams daily
  • Fluid restriction: <2 liters daily (more restrictive in severe hyponatremia)
  • Daily weights: Target body weight; alert if gain >2–3 lbs in 1–2 days
  • Intake/output monitoring: Document all sources
  • Continue guideline-directed medical therapy if blood pressure permits

Congestion ("Wet" Status) — LMNOP Protocol

When pulmonary or peripheral edema is present, use the LMNOP mnemonic:

Intervention Details
Lasix (Furosemide) Start 40–160 mg IV Q12H; typically 2.5x home oral dose. Titrate to euvolemia.
Morphine 2–4 mg IV; improves dyspnea via vasodilation + anxiolysis. Use caution in hypotension/hypoxia.
Nitrates Nitroglycerin IV (goal SBP >100) or isosorbide; improves preload + afterload.
Oxygen Target SpO2 ≥90%; use cautiously to avoid hypercapnia in COPD overlap.
Position Elevate head of bed 30–45°; improves ventilation.

Loop diuretic equivalents (approximate for calculating doses):

Agent Equivalent Dose
Furosemide (Lasix) 20 mg IV = 40 mg PO
Torsemide 20 mg (more potent than furosemide)
Bumetanide 1 mg (most potent, short duration)

Diuretic Dosing Pitfall

Do not use the same IV dose as home PO dose. IV furosemide is approximately 2.5 times more potent than oral.

Afterload Reduction

When systolic dysfunction is present without hypotension:

  • ACE inhibitor (e.g., lisinopril) or ARB (e.g., losartan)
  • Hydralazine + isosorbide dinitrate combination — particularly beneficial in African Americans
  • IV nitroglycerin — for acute afterload reduction

Cardiogenic Shock Management

  • ICU-level care required
  • Inotropic support:
  • Dobutamine 2–20 mcg/kg/min (increases contractility + decreases afterload)
  • Milrinone 0.25–0.75 mcg/kg/min (phosphodiesterase-3 inhibitor; inotrope + vasodilator)
  • Vasopressors: Norepinephrine or dopamine if hypotensive despite inotropes
  • Mechanical support: Intra-aortic balloon pump, percutaneous ventricular assist device, extracorporeal membrane oxygenation (ECMO)
  • Renal replacement therapy: Consider if oliguria develops
  • Urgent cardiology/transplant consultation

Documentation in the Medical Record

Every HF encounter should clearly document:

  • Current NYHA functional class
  • Type of HF (systolic vs. diastolic, ischemic vs. nonischemic)
  • Most recent ejection fraction and date of last echocardiogram
  • Dry weight (helps guide diuretic dosing on future visits)
  • Current medication regimen with doses
  • Renal function and electrolytes from most recent labs
  • Volume status assessment (wet vs. dry)

Atrial Fibrillation: Diagnosis, Risk Stratification, and Management

Classification of Atrial Fibrillation

Category Duration Clinical Notes
Paroxysmal <48 hours; self-terminates May progress to persistent AF over time
Persistent >7 days; requires intervention for termination Sustained but not permanent
Long-standing persistent >1 year on anticoagulation Chronicity increases structural remodeling
Permanent Accepted by patient and provider as indefinite Reversion not pursued

Additionally, classify as valvular (structural valve disease or prosthetic) or nonvalvular (all other cases), as this affects anticoagulation choice.

Etiology of Atrial Fibrillation

Identifying the Trigger

Finding and treating the underlying cause is as important as rate/rhythm control.

Cardiac causes: - Heart failure (systolic or diastolic) - Cardiomyopathy (ischemic or nonischemic) - Myocardial infarction or active ischemia - Valvular disease (mitral stenosis particularly) - Atrial myxoma or other structural abnormality

Pulmonary causes: - COPD or other chronic lung disease - Pneumonia or acute respiratory infection - Pulmonary embolism - Obstructive sleep apnea

Metabolic/Endocrine causes: - Thyrotoxicosis or hyperthyroidism - Elevated catecholamines (pheochromocytoma)

Exogenous triggers: - Alcohol (chronic heavy use or acute binge) - Cocaine or other stimulants - Caffeine excess - Theophylline

Neurogenic causes: - Subarachnoid hemorrhage - Acute stroke - Traumatic brain injury

Acute Rate Control

When the patient is hemodynamically stable and rapid ventricular response requires rate control, use IV agents:

Agent IV Dosing Mechanism Notes
Verapamil 5–10 mg IV bolus; may repeat in 15 min Calcium channel blocker Fast onset; contraindicated if hypotensive or heart failure
Diltiazem 0.25 mg/kg IV bolus; repeat if needed Calcium channel blocker Alternative to verapamil with less negative inotropy
Metoprolol 5–15 mg IV Q6H Beta-blocker Slower onset than IV calcium blockers
Esmolol 0.5 mg/kg bolus, then infusion Ultra-short-acting beta-blocker Use when rapid reversibility desired
Digoxin 0.5 mg IV, then 0.25 mg Q6H Vagomimetic + AV nodal blocker Narrow therapeutic window; less preferred acutely
Amiodarone 150 mg IV over 10 min, then infusion Antiarrhythmic (class III) Last-line if others contraindicated or ineffective

Maintenance Rate Control

Outpatient oral agents for rhythm control:

  • Beta-blockers (metoprolol ER, atenolol, carvedilol)
  • Calcium channel blockers (diltiazem ER, verapamil ER)
  • Digoxin (if significant HFrEF or sedentary lifestyle)

Target heart rate depends on symptoms and function:

Scenario Target HR
Symptomatic AF with EF >40% <80 bpm at rest; <110 with moderate activity
Asymptomatic AF with EF >40% <110 bpm (lenient rate control) acceptable
AF with HFrEF (EF <40%) <80 bpm preferred

Stroke Risk Stratification: CHA₂DS₂-VASc Score

This score predicts stroke risk and guides anticoagulation intensity:

Risk Factor Points
Congestive heart failure or LV dysfunction 1
Hypertension (on treatment) 1
Age ≥75 years 2
Diabetes mellitus 1
Stroke/TIA/thromboembolism history 2
Vascular disease (MI, peripheral artery disease, aortic plaque) 1
Age 65–74 years 1
Sex category (female) 1

Anticoagulation recommendations:

Score Recommendation
0 (male) or 1 (female) No anticoagulation; consider aspirin 75–325 mg daily
1 (male) or 2 (female) Anticoagulation or aspirin (shared decision-making)
≥2 (male) or ≥3 (female) Oral anticoagulation indicated

Bleeding Risk: HAS-BLED Score

Identifies patients at higher bleeding risk while on anticoagulation:

Risk Factor Points
Hypertension (uncontrolled) 1
Abnormal renal/liver function 1
Stroke history 1
Bleeding history 1
Labile INR (if on warfarin) 1
Elderly (age >65) 1
Drug use (NSAIDs, antiplatelet) or alcohol excess 1

Interpretation:

  • Score <3: Low bleeding risk
  • Score ≥3: Assess risk vs. benefit; do not withhold anticoagulation, but monitor closely

Anticoagulation Options

Anticoagulation is Essential

Most AF patients with CHA₂DS₂-VASc ≥2 require anticoagulation to reduce stroke risk by ~60%.

Agent Dosing Monitoring Key Advantages Limitations
Dabigatran 150 mg BID (110 mg BID if high bleeding risk) No routine labs; check renal function baseline Rapid onset/offset; predictable PK Dyspepsia; GI bleed risk; must take intact
Rivaroxaban 20 mg daily with food (15 mg if CrCl 15–30) No routine labs; check renal function baseline Once daily; can open capsule if needed Food interaction; GI bleed risk
Apixaban 5 mg BID (2.5 mg BID if ≥2 of: age ≥60, weight ≤60 kg, Cr ≥1.5) No routine labs; check renal function baseline Lowest GI bleed risk; twice-daily convenient for some Twice-daily dosing
Edoxaban 60 mg daily (30 mg if weight <60 kg, CrCl 15–50, or concurrent strong P-gp inhibitor) No routine labs Once daily More strokes if CrCl >95; less data in valvular AF
Warfarin Dose adjusted for INR 2–3 INR monitoring (initially frequent, then q4 weeks) Decades of safety data; reversible with vitamin K Dietary interactions; narrow therapeutic window; frequent monitoring
Aspirin 75–325 mg daily No monitoring Readily available Inferior efficacy vs. anticoagulants; used only if AC contraindicated


Last update: April 20, 2026